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Trans-FA inhibits the proliferation of H1299 lung cancer cells and induces a average increase in the apoptotic populace by promoting phosphorylation of β-catenin at residues Thr41 and Ser45 and resulting in proteasomal degradation [117,177]. Intriguingly, FA is capable of inhibiting the proliferation and migration of lung cancer cells by eliminati

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being an autophagy inducer to show the utility of p62 and LC3B-II quantification in HEK293T cells and primary cultures of rat neurons and astrocytes applying time-settled fluorescence resonance Strength transfer (TR-FRET)C. The corporate reserves the best, but will not presume any obligation, to (1) clear away any substance posted on the internet W

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